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WHY ARE SVR, SV AND SVV OF INTEREST

Function of arteries

1) Conduits by which means an adequate supply of blood is delivered to body tissues

2) Cushions the action of the pulse resulting from intermittent ventricular ejection are dampened (thus affects pulse pressure and pulsatile flow) Mean arterial pressure (MAP) = CO x SVR = ~ peak of curve

CO (Cardiac Output) = height of overall curve in combination with gain SVR = height of PreSystolic Amplitudes (PSA) in combination with gain and in relation to CO

Cushion function: instantaneous accommodation of blood ejected from the ventricles, storing part of the stroke volume during systole and draining this volume during diastole, thus permitting continuous perfusion of peripheral organs and tissues. Influenced by

• ventricular ejection (SV),
• arterial stiffness,
• wave reflections

See how arterial elasticity can be assessed with the MDSoftware available with the vet hdo equipment

PWA enables the practitioner to look for:

1) Stress related changes: higher PSA due to epinephrine / norepinephrine effects on arteries (increased SVR), whereas SVV (alternating high and low single amplitudes) relates to the consequent effects on the myocardium.

2) True hypertension (very high PSA = clearly affected SVR)

3) Stability of systolic function (SV, SVV)

4) Severity of effect on cardiac output due to mitral regurgitation, dilated and hypertrophic cardiomyopathy (SV, SVV)

5) Severity of arrhythmias (SVV)

6) Activation of RAAS (PSA/SVR), supporting
– Early diagnosis of kidney disease
– Early diagnosis of heart disease if no Echo is available
– Treatment decision on ACE-Inhibitor and other drugs

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